EMF Block Paint plus EMF Shielding

· 2 min read
EMF Block Paint plus EMF Shielding

Whether you live in a house or apartment or just need to ensure that your house is free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the most effective is to restrict the use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from entering your home. Another easy way to protect your house against EMF radiations is to install an RF shielding canopy. It's a kind of net that has EMF shielding. It is utilized to prevent EMFs from entering rooms. Another option is to get your house equipped with an electrical enclosure.  https://paste1s.com/notes/W5VHZW  are called Faraday cages.

Several studies have shown how the EMF that is not ionized produces antiproliferative effects in HCC cells.  https://fnote.me/notes/Wb28zr  behind AM RF EMF's anticancer activity in vitro is believed result from the deregulation of cancer stem cells. This could be the reason for the long-term response seen in some patients with advanced HCC. But,  blocking emf  for AM EMF's impact on cancer patients isn't yet clear.

Aspects from AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were examined in mice. The tumours were classified in three different groups. One group did not have exposure RF EMF. Second group members were exposed to RF EMF at frequencies similar to that of humans. The third group was exposed RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on the tumours was assessed against the effect of RCF. The results indicated that tumors treated by HCCMF were significantly shrinking. However, the tumours treated with RCF didn't show evidence of tumour shrinkage.


The reason for tumor-specific AM RF EMF might be driven by the fact that tumour cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is controlled through CACNA1H, a protein that mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H may have broader implications in the diagnosis and treatment of various cancers.

The tumors in the controls were never exposed to EMF from RF, and fed a standard mouse diet. The tumours in those in the HCCMF group were infected with Huh7 cells at the time they were five-seven weeks old. The tumours were then euthanized after they had a high burden.

The tumors of the three groups also displayed distinct growth curves. The HCCMF-treated tumors saw a significant decrease in size of the tumor after 8 weeks. However, the tumours treated with RCF didn't show reduction in size. The difference was significant. The tumors treated with RCF showed necrosis that is common in tumours exposed to RCF. It is possible that the necrosis was due to an absence of oxygen in the more invasive tumours.

In sum, the results show that AM RF EMF has anti-cancer properties in vitro as well as in the vivo. Numerous studies have demonstrated it is true that AM RF EMF produces measurable tumour shrinkage within HCC patients. There is a possibility that AM RF EMF produces these effects through CACNA1H which is a protein involved in the tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained influence on the growth of HCC tumours in living tissue.